Increasing temperatures are of major concern worldwide as it results in
decreased crop yield if plants cannot acclimate to this stress. Furthermore,
not clear as to how it effects on other diseases such as pathogen attacks by
making plants even more vulnerable.
Ascochyta blight by
Didymella pinodes, is a main cause for severe losses of pea
production every year. Therefor, the search for resistance traits and their exact regulation is a major
goal for farmers.
Aim: By focusing on target metabolic pathways, we want to better understand signal transduction and turnover dynamics
for the early defense mechanisms of different Pisum sativum cultivars upon pathogen attack and how these
are affected by heat stress.
This project is based on a prevously fundet FWF Project [P24870-B22]:
Tripartite symbiosis formed by Pisum sativum, rhizobia and mycorrhiza...more »
Target proteins and pathways are based on those works, where we found
that microsymbions induced systemic resistance upon D. pinodes in P.
sativum by enhanced levels of several defense mechanisms prior and during
pathogen attack. Cultivars molecular background can further promote these
1) The pisatin pathway is involved in early pathogen defense. It hampers the
pathogen switching from the biotrophic- to necrotrophic state. What is the
turnover rate of that pathways
proteome from onset of infection? Does reaction time and/or intensity matter? How?
2) The monolignol pathway regulation upon pathogen response is an interplay between different
compartments. How/when and where are the enzymes of this pathway synthesized during infection?
3) Increasing temperatures will affect pathogenicity. How?
Stefanie Wienkoop »